neuromuscular junctions การใช้

• It occurs in neuromuscular junction disorders, such as myasthenia gravis.
• WNT4 contributes to the formation of the neuromuscular junction in vertebrates.
• LRP4 plays an important role in the formation of neuromuscular junctions.
• BTX-A inhibits release of acetylcholine acting at the neuromuscular junction.
• The neurotoxin prevents presynaptic release of acetylcholine from neuromuscular junctions.
• This connection between motor neurons and muscles is called the neuromuscular junction.
• Non-quantal and quantal release of acetylcholine at the neuromuscular junction.
• The neuromuscular junction is the site of the signal exchange.
• The two cysteine residues form a glutamatergic neuromuscular junction.
• Neuromuscular junction of the rat, mouse and frog.
• They function by propagating graded potentials and serve to modulate some neuromuscular junctions.
• Botulinum toxin causes temporary muscular paralysis by inhibiting acetylcholine release at the neuromuscular junction.
• Neuromuscular junction diseases can be of autoimmune origin.
• Acetylcholine concentrations then increase at neuromuscular junctions to cause involuntary contraction of all muscles.
• This application enables morphological characterization of neuromuscular junctions.
• Botox selectively blocks muscle feedback by blocking presynaptic acetylcholine receptors at the neuromuscular junction.
• It is located in the neuromuscular junction, where activation yields K + conductance.
• Neuromuscular junction diseases can also be referred to as end plate diseases or disorders.
• Neuromuscular junction : the messenger axon cell tells muscle cells to contract at this intersection
• Botulinum toxin causes muscle paralysis / atrophy by inhibition of acetylcholine release at neuromuscular junctions.